2
Peer #1
Cleveland Clinic. (2022). Malnutrition. Retrieved from
Keller, U. (2019). Nutritional Laboratory Markers in Malnutrition. Journal of Clinical Medicine, 8(6), 775.
McCance, K. L., & Huether, S. E. (2019). Pathophysiology: The biological basis for disease in adults and
children (8th ed.). St. Louis, MO: Mosby/Elsevier.
MEDLINE Plus. (n.d.). Albumin Blood Test. Retrieved from
Peer #2
In scenario 4 the patient is a 27-year-old who appears to be suffering from a myocardial infarction possibly because of prolonged drug use. Myocardial infarction (MI), colloquially known as "heart attack," is caused by decreased or complete cessation. It is possible that ion of blood flow to a portion of the myocardium ( Dhamoon). Although our patient was only 27 years old if he has a familial history of myocardial infarction, A systematic comparison of these markers in thousands of patients with myocardial infarction and in healthy control subjects has enabled the identification of gene segments whose influence on the risk of atherosclerosis is established in beyond any doubt (Erdmann). Some other risk factors of heart attack are smoking, obesity, high blood pressure, hypercholesteremia, and age. The main cells affected are blood and arteries. The build up of plaque in arteries preventing adequate blood flow to remainder of body. In the scenario this explains how the patient’s potassium was 6.9 meq/l resulting in the abnormal EKG results. The buildup caused the necrotic tissue causing burning sensation over hip and forearm. The necrotic or dead tissue is a result of decreased blood flow to area preventing cell revitalization. While the article doesn’t state what type of drug it is implied patient is taking a central nervous system stimulants and other drugs that have the potential for myocardial damage (Bergstrom). There are no changes in heart attack in women studies show There is less data regarding treatment modalities based upon gender differences but in general, it appears that women benefit from established treatment modalities in the acute setting and after the event. It has been determined that there are epidemiologic differences, but pathophysiologic and clinical factors specific to women in this setting have yet to be determined (Mendelsohn). my responses would not change except medical staff would need to be more vigilant with symptom monitoring in order to diagnose Myocardial Infarction in women as the signs are often missed.
Bergstrom, D. L., & Keller, C. (1992). Drug-induced myocardial ischemia and acute myocardial infarction. Critical care nursing clinics of North America, 4(2), 273–278.
Erdmann, J., Linsel-Nitschke, P., & Schunkert, H. (2010). Genetic causes of myocardial infarction: new insights from genome-wide association studies. Deutsches Arzteblatt international, 107(40), 694–699. https://doi.org/10.3238/arztebl.2010.0694
Mendelson, M. A., & Hendel, R. C. (1995). Myocardial infarction in women. Cardiology, 86(4), 272–285. https://doi.org/10.1159/000176891
Ojha N, Dhamoon AS. Myocardial Infarction. [Updated 2022 Aug 8]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2022 Jan-. Available from: https://www.ncbi.nlm.nih.gov/books/NBK537076/
